A new study is now suggesting that sleeping with the blinds open could raise your risk of stroke or heart attack by 43 percent.
That’s right, experts claim the massive surge in heart attacks and strokes has nothing to do with the vaccine rollout, but rather to do with street lights and light pollution from traffic disrupting the sleep-wake cycle.
Researchers in China have now compared data from thousands of people, concluding that sleeping with your blinds open is suddenly a major health risk.
Dr Jain-Bing Wang, a health expert involved in the paper, said:
“Our study suggests that higher levels of exposure to outdoor artificial light at night may be a risk factor for cerebrovascular disease.”
“Therefore, we advise people, especially those living in urban areas, to consider reducing that exposure to protect themselves from its potential harmful impact.”
Scientists now warn that 80 percent of the world’s population lives in light-polluted areas, which pose significant health risks.
According to the experts, middle-aged women who sleep less than five hours every night are 75 percent more likely to suffer heart failure or stroke.
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The Daily Mail reported:
Using the Bortle Dark-Sky Scale, which rates the darkness of the sky from one to nine, scientists say the average moonless night has a measurement of one — or very little light in the sky.
For comparison, major cities like New York have a measurement of nine — suggesting high levels of light pollution.
For the paper, published today in the journal Stroke, scientists recruited 28,300 people from the Chinese port city of Ningbo, about 120 miles South of Shanghai.
Participants were 62 years old on average and did not have a history of cerebrovascular disease — such as stroke and aneurysms.
Each was tracked for six years — from 2015 to 2021 — with scientists recording cases of stroke or other cerebrovascular disease via hospital medical records.
They detected 1,278 cases overall, of which 900 were strokes.
For the analysis, satellite images were used to estimate the light pollution each patient suffered from at night.
Results were adjusted by age, sex, and income to suggest the risk of suffering cerebrovascular disease from light exposure at night.
In the paper, the researchers wrote:
“Exposure to [bright light at night] could lead to the body’s circadian rhythm suppressing melatonin secretion.”
“This could result in changes in biological indicators including elevated triglyceride levels… blood pressure and blood glucose, which are all triggers for the occurrence of cerebrovascular disease.”
Dr Wang added:
“Despite significant advances in reducing traditional cardiovascular risk factors such as smoking, obesity, and type 2 diabetes, it is important to consider environmental factors in our efforts to decrease the global burden of cardiovascular disease.”
The limitations of the paper included the fact that there was no data on indoor light exposure for participants or whether they used blackout curtains.
The research was also carried out partly over the Covid pandemic, which may have affected people’s disease risk via lifestyle changes.
The CDC recommends that everyone sleep between seven and nine hours every night.
The study also investigated the risk of cerebrovascular disease from air pollution.
It found that people living in areas with the highest levels of PM10 — a tiny compound released from car exhausts—had up to a 50 percent higher risk of suffering from cerebrovascular disease than those exposed to the lowest levels.
Those exposed to the highest levels of PM2.5 — a smaller substance also released from cars — had a 41 percent higher risk.
And those exposed to nitrogen oxide, released from cars and industrial areas, had a 31 percent higher risk.
The researchers said there was no link between air pollution and light pollution and the risk of cerebrovascular disease.
Explaining the higher risk from air pollution in the paper, they added:
“An elevated risk of hemorrhagic stroke may be achieved by provoking arterial vasoconstriction, elevated blood pressure, and increased susceptibility to cerebral vascular rupture by triggering endothelial dysfunction.”
